Inhibition of glycogen synthase kinase-3 activity triggers an apoptotic response in pancreatic cancer cells through JNK-dependent mechanisms. [electronic resource]
Producer: 20120508Description: 529-37 p. digitalISSN:- 1460-2180
- Anthracenes -- pharmacology
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins -- biosynthesis
- Bcl-2-Like Protein 11
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- Cell Survival -- drug effects
- Glycogen Synthase Kinase 3 -- antagonists & inhibitors
- Humans
- Indoles -- pharmacology
- JNK Mitogen-Activated Protein Kinases -- metabolism
- MAP Kinase Signaling System
- Maleimides -- pharmacology
- Membrane Proteins -- biosynthesis
- Pancreas -- metabolism
- Pancreatic Neoplasms -- metabolism
- Phosphorylation
- Proto-Oncogene Proteins -- biosynthesis
- Proto-Oncogene Proteins c-akt -- metabolism
- Pyridines -- pharmacology
- Pyrimidines -- pharmacology
- RNA, Messenger -- biosynthesis
- Receptors, Death Domain -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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