Turmeric (Curcuma longa) inhibits inflammatory nuclear factor (NF)-κB and NF-κB-regulated gene products and induces death receptors leading to suppressed proliferation, induced chemosensitization, and suppressed osteoclastogenesis. [electronic resource]
Producer: 20120726Description: 454-65 p. digitalISSN:- 1613-4133
- Animals
- Anti-Inflammatory Agents -- pharmacology
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Blotting, Western
- Capecitabine
- Cell Differentiation -- drug effects
- Cell Proliferation -- drug effects
- Curcuma -- chemistry
- Cyclin D1 -- genetics
- Deoxycytidine -- analogs & derivatives
- Down-Regulation
- Fluorouracil -- analogs & derivatives
- HCT116 Cells
- HT29 Cells
- Humans
- Mice
- NF-kappa B -- antagonists & inhibitors
- Osteoclasts -- cytology
- Paclitaxel -- pharmacology
- Plant Extracts -- pharmacology
- RANK Ligand -- genetics
- Reactive Oxygen Species -- metabolism
- Receptors, CXCR4 -- genetics
- Receptors, Death Domain -- genetics
- STAT3 Transcription Factor -- genetics
- X-Linked Inhibitor of Apoptosis Protein -- genetics
- bcl-2-Associated X Protein -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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