Spinal matrix metalloproteinase 3 mediates inflammatory hyperalgesia via a tumor necrosis factor-dependent mechanism. [electronic resource]
Producer: 20120507Description: 199-210 p. digitalISSN:- 1873-7544
- Analysis of Variance
- Animals
- Anti-Inflammatory Agents, Non-Steroidal -- therapeutic use
- Calcium-Binding Proteins
- Cells, Cultured
- DNA-Binding Proteins -- metabolism
- Dipeptides -- therapeutic use
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Enzyme Inhibitors -- therapeutic use
- Enzyme-Linked Immunosorbent Assay -- methods
- Etanercept
- Gene Expression Regulation -- drug effects
- Glial Fibrillary Acidic Protein -- metabolism
- Hydroxamic Acids -- therapeutic use
- Hyperalgesia -- drug therapy
- Immunoglobulin G -- therapeutic use
- Inflammation -- chemically induced
- Lipopolysaccharides
- Male
- Matrix Metalloproteinase 3 -- administration & dosage
- Microfilament Proteins
- Neuroglia -- drug effects
- Pain Measurement
- Pain Threshold -- drug effects
- Physical Stimulation
- Rats
- Rats, Sprague-Dawley
- Reaction Time -- drug effects
- Receptors, Tumor Necrosis Factor -- therapeutic use
- Spinal Cord -- drug effects
- Sulfonamides -- therapeutic use
- Time Factors
- Tumor Necrosis Factors -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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