PGE(2)-induced CXCL12 production and CXCR4 expression controls the accumulation of human MDSCs in ovarian cancer environment. [electronic resource]
Producer: 20120306Description: 7463-70 p. digitalISSN:- 1538-7445
- Biphenyl Compounds -- pharmacology
- CD11b Antigen -- genetics
- Celecoxib
- Cell Movement
- Cells, Cultured
- Chemokine CXCL12 -- genetics
- Cyclooxygenase 2 -- genetics
- Cyclooxygenase 2 Inhibitors -- pharmacology
- Dinoprostone -- metabolism
- Female
- Flow Cytometry
- Gene Expression Regulation, Neoplastic
- Humans
- Monocytes -- drug effects
- Myeloid Cells -- drug effects
- Ovarian Neoplasms -- genetics
- Prostaglandin Antagonists -- pharmacology
- Pyrazoles -- pharmacology
- Receptors, CXCR4 -- genetics
- Receptors, Prostaglandin E, EP2 Subtype -- antagonists & inhibitors
- Receptors, Prostaglandin E, EP4 Subtype -- antagonists & inhibitors
- Sulfonamides -- pharmacology
- Tumor Cells, Cultured
- Tumor Microenvironment -- drug effects
- Xanthones -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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