Orthovanadate-induced cell death in RET/PTC1-harboring cancer cells involves the activation of caspases and altered signaling through PI3K/Akt/mTOR. [electronic resource]
Producer: 20111012Description: 371-7 p. digitalISSN:- 1879-0631
- Antineoplastic Agents -- pharmacology
- Apoptosis
- Carcinoma
- Carcinoma, Papillary
- Caspases -- metabolism
- Cell Proliferation -- drug effects
- Enzyme Activation
- Glial Cell Line-Derived Neurotrophic Factor -- metabolism
- Humans
- Membrane Potential, Mitochondrial -- drug effects
- Oncogene Protein v-akt -- metabolism
- Oncogene Proteins, Fusion -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphorylation
- Protein-Tyrosine Kinases -- antagonists & inhibitors
- Reactive Oxygen Species -- metabolism
- Signal Transduction
- TOR Serine-Threonine Kinases -- metabolism
- Thyroid Cancer, Papillary
- Thyroid Neoplasms -- genetics
- Tumor Cells, Cultured
- Vanadates -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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