Prolactin increases SMN expression and survival in a mouse model of severe spinal muscular atrophy via the STAT5 pathway. [electronic resource]
Producer: 20111003Description: 3042-50 p. digitalISSN:- 1558-8238
- Animals
- Disease Models, Animal
- Endothelial Cells -- cytology
- Gene Expression Regulation
- Humans
- Mice
- Motor Neurons -- physiology
- Muscular Atrophy, Spinal -- metabolism
- Neurons -- metabolism
- Prolactin -- biosynthesis
- RNA, Messenger -- metabolism
- STAT5 Transcription Factor -- metabolism
- Survival of Motor Neuron 1 Protein -- biosynthesis
- Transcription, Genetic
- Tumor Suppressor Proteins -- metabolism
- Up-Regulation
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.