Dual blockade of EGFR and c-Met abrogates redundant signaling and proliferation in head and neck carcinoma cells. [electronic resource]
Producer: 20111024Description: 4425-38 p. digitalISSN:- 1557-3265
- Animals
- Antineoplastic Agents -- pharmacology
- Carcinoma -- metabolism
- Carcinoma, Squamous Cell
- Cell Line, Tumor
- Cell Proliferation -- drug effects
- ErbB Receptors -- antagonists & inhibitors
- Female
- Gefitinib
- Head and Neck Neoplasms -- metabolism
- Hepatocyte Growth Factor -- metabolism
- Humans
- Mice
- Mice, SCID
- Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Neoplasm Invasiveness
- Neoplasms, Squamous Cell -- metabolism
- Phosphorylation -- drug effects
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Proto-Oncogene Proteins c-met -- antagonists & inhibitors
- Quinazolines -- pharmacology
- Signal Transduction -- drug effects
- Squamous Cell Carcinoma of Head and Neck
- Transforming Growth Factor alpha -- pharmacology
- Wound Healing -- drug effects
- Xenograft Model Antitumor Assays
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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