Spinal injection of TNF-α-activated astrocytes produces persistent pain symptom mechanical allodynia by releasing monocyte chemoattractant protein-1. [electronic resource]
Producer: 20110126Description: 1871-80 p. digitalISSN:- 1098-1136
- Animals
- Animals, Newborn
- Antibodies -- therapeutic use
- Astrocytes -- drug effects
- Calcium-Binding Proteins -- metabolism
- Cells, Cultured
- Cerebral Cortex -- cytology
- Chemokine CCL2 -- immunology
- Disease Models, Animal
- Enzyme Inhibitors -- pharmacology
- Enzyme-Linked Immunosorbent Assay -- methods
- Gene Expression Regulation -- drug effects
- Glial Fibrillary Acidic Protein -- metabolism
- Hyperalgesia -- chemically induced
- Injections, Spinal -- methods
- MAP Kinase Kinase 4 -- metabolism
- Male
- Mice
- Mice, Knockout
- Microfilament Proteins
- Pain Threshold -- drug effects
- RNA, Small Interfering -- therapeutic use
- Receptors, Tumor Necrosis Factor -- deficiency
- Receptors, Tumor Necrosis Factor, Type I -- deficiency
- Spinal Cord -- pathology
- Tumor Necrosis Factor-alpha -- adverse effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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