Poly(I:C) drives type I IFN- and TGFβ-mediated inflammation and dermal fibrosis simulating altered gene expression in systemic sclerosis. [electronic resource]
Producer: 20101116Description: 2583-93 p. digitalISSN:- 1523-1747
- Adaptor Proteins, Vesicular Transport -- genetics
- Animals
- Cells, Cultured
- Dermatitis -- genetics
- Dermis -- immunology
- Disease Models, Animal
- Fibroblasts -- cytology
- GTP-Binding Proteins -- metabolism
- Gene Expression -- immunology
- Humans
- Interferon Type I -- immunology
- Interferon-gamma -- immunology
- Lupus Erythematosus, Cutaneous -- genetics
- Mice
- Mice, Inbred C57BL
- Mice, Mutant Strains
- Myxovirus Resistance Proteins
- Poly I-C -- metabolism
- Scleroderma, Systemic -- genetics
- Toll-Like Receptor 3 -- agonists
- Transforming Growth Factor beta -- immunology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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