Decreased transcription-coupled nucleotide excision repair capacity is associated with increased p53- and MLH1-independent apoptosis in response to cisplatin. [electronic resource]
Producer: 20100816Description: 207 p. digitalISSN:- 1471-2407
- Adaptor Proteins, Signal Transducing -- genetics
- Antineoplastic Agents -- pharmacology
- Apoptosis -- drug effects
- Cisplatin -- pharmacology
- Colorectal Neoplasms -- enzymology
- DNA Helicases -- genetics
- DNA Repair -- radiation effects
- DNA Repair Enzymes -- genetics
- Dose-Response Relationship, Drug
- Dose-Response Relationship, Radiation
- Down-Regulation
- Drug Resistance, Neoplasm
- Gene Expression Regulation, Neoplastic
- HCT116 Cells
- Humans
- Male
- MutL Protein Homolog 1
- Nuclear Proteins -- genetics
- Poly-ADP-Ribose Binding Proteins
- Prostatic Neoplasms -- enzymology
- RNA Interference
- Transcription, Genetic
- Tumor Suppressor Protein p53 -- genetics
- Ultraviolet Rays
- Up-Regulation
- Xeroderma Pigmentosum Group A Protein -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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