Mice deficient in Mkp-1 develop more severe pulmonary hypertension and greater lung protein levels of arginase in response to chronic hypoxia. [electronic resource]
Producer: 20100518Description: H1518-28 p. digitalISSN:- 1522-1539
- Animals
- Arginase -- metabolism
- Blotting, Western
- Body Weight -- physiology
- Chronic Disease
- Dual Specificity Phosphatase 1 -- genetics
- Hypertension, Pulmonary -- genetics
- Hypoxia -- genetics
- Lung -- metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardium -- metabolism
- Nitrates -- metabolism
- Nitric Oxide Synthase Type I -- biosynthesis
- Nitric Oxide Synthase Type II -- biosynthesis
- Nitric Oxide Synthase Type III -- biosynthesis
- Nitrites -- metabolism
- Proliferating Cell Nuclear Antigen -- metabolism
- Proteins -- metabolism
- Ventricular Function, Right -- physiology
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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