Induction of p53 renders ATM-deficient mice refractory to hepatocarcinogenesis. [electronic resource]
Producer: 20100325Description: 1155-65.e1-2 p. digitalISSN:- 1528-0012
- Animals
- Apoptosis
- Ataxia Telangiectasia Mutated Proteins
- Carcinoma, Hepatocellular -- chemically induced
- Caspase 3 -- metabolism
- Cell Cycle
- Cell Cycle Proteins -- genetics
- Cell Transformation, Neoplastic -- chemically induced
- Cellular Senescence
- Checkpoint Kinase 1
- Chromosome Aberrations
- Cyclin-Dependent Kinase Inhibitor p16 -- metabolism
- DNA-Binding Proteins -- deficiency
- Diethylnitrosamine
- Disease Models, Animal
- Gene Expression Regulation, Neoplastic
- Heterozygote
- Karyotyping
- Liver Neoplasms -- chemically induced
- Male
- Mice
- Mice, Knockout
- Protein Kinases -- metabolism
- Protein Serine-Threonine Kinases -- deficiency
- Signal Transduction
- Telomerase -- metabolism
- Time Factors
- Tumor Suppressor Protein p53 -- genetics
- Tumor Suppressor Proteins -- deficiency
- Up-Regulation
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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