Amyloid beta-derived neuroplasticity in bone marrow-derived mesenchymal stem cells is mediated by NPY and 5-HT2B receptors via ERK1/2 signalling pathways. [electronic resource]
Producer: 20091008Description: 571-86 p. digitalISSN:- 1365-2184
- Alzheimer Disease -- metabolism
- Amyloid beta-Peptides -- metabolism
- Animals
- Apoptosis -- drug effects
- Bone Marrow Cells -- cytology
- Cell Differentiation -- drug effects
- Cell Division -- drug effects
- Cell Survival -- drug effects
- Cells, Cultured
- Dynamins -- metabolism
- MAP Kinase Signaling System -- physiology
- Mesenchymal Stem Cells -- cytology
- Mice
- Mice, Inbred C57BL
- Mice, Mutant Strains
- Mitogen-Activated Protein Kinase 1 -- metabolism
- Mitogen-Activated Protein Kinase 3 -- metabolism
- Neuronal Plasticity -- physiology
- Neuropeptide Y -- genetics
- Peptide Fragments -- metabolism
- Phosphatidylinositol 3-Kinases -- metabolism
- Receptor, Serotonin, 5-HT2B -- metabolism
- Receptors, Neuropeptide Y -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.