Amyloid beta-derived neuroplasticity in bone marrow-derived mesenchymal stem cells is mediated by NPY and 5-HT2B receptors via ERK1/2 signalling pathways.

Jin, H K

Amyloid beta-derived neuroplasticity in bone marrow-derived mesenchymal stem cells is mediated by NPY and 5-HT2B receptors via ERK1/2 signalling pathways. [electronic resource] - Cell proliferation Oct 2009 - 571-86 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1365-2184

10.1111/j.1365-2184.2009.00625.x doi


Alzheimer Disease--metabolism
Amyloid beta-Peptides--metabolism
Animals
Apoptosis--drug effects
Bone Marrow Cells--cytology
Cell Differentiation--drug effects
Cell Division--drug effects
Cell Survival--drug effects
Cells, Cultured
Dynamins--metabolism
MAP Kinase Signaling System--physiology
Mesenchymal Stem Cells--cytology
Mice
Mice, Inbred C57BL
Mice, Mutant Strains
Mitogen-Activated Protein Kinase 1--metabolism
Mitogen-Activated Protein Kinase 3--metabolism
Neuronal Plasticity--physiology
Neuropeptide Y--genetics
Peptide Fragments--metabolism
Phosphatidylinositol 3-Kinases--metabolism
Receptor, Serotonin, 5-HT2B--metabolism
Receptors, Neuropeptide Y--metabolism