Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury. [electronic resource]
Producer: 20090610Description: H1125-32 p. digitalISSN:- 0363-6135
- Animals
- Calcium -- metabolism
- Cyclosporine -- pharmacology
- Fluoresceins -- metabolism
- Hydrogen Peroxide -- pharmacology
- Magnetic Resonance Spectroscopy
- Male
- Membrane Potential, Mitochondrial -- drug effects
- Mitochondria, Heart -- drug effects
- Mitochondrial Membrane Transport Proteins -- antagonists & inhibitors
- Mitochondrial Permeability Transition Pore
- Myocardial Contraction -- drug effects
- Myocardium -- cytology
- Myocytes, Cardiac -- cytology
- Rats
- Rats, Sprague-Dawley
- Reactive Oxygen Species -- pharmacology
- Reperfusion Injury -- metabolism
- Ventricular Function, Left -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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