Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury.

Saotome, Masao

Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury. [electronic resource] - American journal of physiology. Heart and circulatory physiology Apr 2009 - H1125-32 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

0363-6135

10.1152/ajpheart.00436.2008 doi


Animals
Calcium--metabolism
Cyclosporine--pharmacology
Fluoresceins--metabolism
Hydrogen Peroxide--pharmacology
Magnetic Resonance Spectroscopy
Male
Membrane Potential, Mitochondrial--drug effects
Mitochondria, Heart--drug effects
Mitochondrial Membrane Transport Proteins--antagonists & inhibitors
Mitochondrial Permeability Transition Pore
Myocardial Contraction--drug effects
Myocardium--cytology
Myocytes, Cardiac--cytology
Rats
Rats, Sprague-Dawley
Reactive Oxygen Species--pharmacology
Reperfusion Injury--metabolism
Ventricular Function, Left--drug effects