Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining. [electronic resource]
Producer: 20081006Description: 2620-5 p. digitalISSN:- 1550-6606
- Animals
- Ataxia Telangiectasia -- enzymology
- Ataxia Telangiectasia Mutated Proteins
- B-Lymphocytes -- enzymology
- Cell Cycle Proteins -- genetics
- Cell Line
- Cell Line, Transformed
- Cells, Cultured
- DNA Breaks, Double-Stranded
- DNA Damage
- DNA Repair
- DNA-Binding Proteins -- deficiency
- Gene Rearrangement, B-Lymphocyte -- genetics
- Mice
- Mice, Knockout
- Mice, Transgenic
- Protein Serine-Threonine Kinases -- deficiency
- Recombination, Genetic
- Stem Cells -- metabolism
- Tumor Suppressor Proteins -- deficiency
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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