Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining.

Bredemeyer, Andrea L

Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining. [electronic resource] - Journal of immunology (Baltimore, Md. : 1950) Aug 2008 - 2620-5 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1550-6606

10.4049/jimmunol.181.4.2620 doi


Animals
Ataxia Telangiectasia--enzymology
Ataxia Telangiectasia Mutated Proteins
B-Lymphocytes--enzymology
Cell Cycle Proteins--genetics
Cell Line
Cell Line, Transformed
Cells, Cultured
DNA Breaks, Double-Stranded
DNA Damage
DNA Repair
DNA-Binding Proteins--deficiency
Gene Rearrangement, B-Lymphocyte--genetics
Mice
Mice, Knockout
Mice, Transgenic
Protein Serine-Threonine Kinases--deficiency
Recombination, Genetic
Stem Cells--metabolism
Tumor Suppressor Proteins--deficiency