Increased activation-induced cell death of high IFN-gamma-producing T(H)1 cells as a mechanism of T(H)2 predominance in atopic diseases. [electronic resource]
Producer: 20080513Description: 652-658.e1 p. digitalISSN:- 1097-6825
- Adult
- Apoptosis -- immunology
- Caspases -- metabolism
- Cell Differentiation -- immunology
- Enzyme Activation -- immunology
- Fas Ligand Protein -- immunology
- Flow Cytometry
- Humans
- Hypersensitivity, Immediate -- immunology
- Interferon-gamma -- biosynthesis
- Lymphocyte Activation -- immunology
- Receptors, Tumor Necrosis Factor, Type II -- immunology
- T-Lymphocyte Subsets -- cytology
- Th1 Cells -- cytology
- Th2 Cells -- cytology
- fas Receptor -- immunology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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