Increased activation-induced cell death of high IFN-gamma-producing T(H)1 cells as a mechanism of T(H)2 predominance in atopic diseases.
Akkoc, Tunc
Increased activation-induced cell death of high IFN-gamma-producing T(H)1 cells as a mechanism of T(H)2 predominance in atopic diseases. [electronic resource] - The Journal of allergy and clinical immunology Mar 2008 - 652-658.e1 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1097-6825
10.1016/j.jaci.2007.12.1171 doi
Adult
Apoptosis--immunology
Caspases--metabolism
Cell Differentiation--immunology
Enzyme Activation--immunology
Fas Ligand Protein--immunology
Flow Cytometry
Humans
Hypersensitivity, Immediate--immunology
Interferon-gamma--biosynthesis
Lymphocyte Activation--immunology
Receptors, Tumor Necrosis Factor, Type II--immunology
T-Lymphocyte Subsets--cytology
Th1 Cells--cytology
Th2 Cells--cytology
fas Receptor--immunology
Increased activation-induced cell death of high IFN-gamma-producing T(H)1 cells as a mechanism of T(H)2 predominance in atopic diseases. [electronic resource] - The Journal of allergy and clinical immunology Mar 2008 - 652-658.e1 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1097-6825
10.1016/j.jaci.2007.12.1171 doi
Adult
Apoptosis--immunology
Caspases--metabolism
Cell Differentiation--immunology
Enzyme Activation--immunology
Fas Ligand Protein--immunology
Flow Cytometry
Humans
Hypersensitivity, Immediate--immunology
Interferon-gamma--biosynthesis
Lymphocyte Activation--immunology
Receptors, Tumor Necrosis Factor, Type II--immunology
T-Lymphocyte Subsets--cytology
Th1 Cells--cytology
Th2 Cells--cytology
fas Receptor--immunology