Dimemorfan protects rats against ischemic stroke through activation of sigma-1 receptor-mediated mechanisms by decreasing glutamate accumulation. [electronic resource]
Producer: 20080205Description: 558-72 p. digitalISSN:- 1471-4159
- Analysis of Variance
- Animals
- Brain Infarction -- etiology
- Brain Ischemia -- drug therapy
- Chemokine CCL2 -- metabolism
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Gene Expression Regulation -- drug effects
- Glutamic Acid -- metabolism
- Lipid Peroxidation -- drug effects
- Male
- Morphinans -- therapeutic use
- Morpholines -- pharmacology
- Nitric Oxide Synthase -- metabolism
- Peroxidase -- metabolism
- Protein Serine-Threonine Kinases -- metabolism
- Rats
- Rats, Long-Evans
- Receptors, sigma -- agonists
- Reperfusion Injury -- prevention & control
- STAT1 Transcription Factor -- metabolism
- Signal Transduction -- drug effects
- Sigma-1 Receptor
- NF-kappaB-Inducing Kinase
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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