Inactivation of glycogen synthase kinase-3beta contributes to brain-derived neutrophic factor/TrkB-induced resistance to chemotherapy in neuroblastoma cells. [electronic resource]
Producer: 20080319Description: 3113-21 p. digitalISSN:- 1535-7163
- Antineoplastic Agents -- therapeutic use
- Brain-Derived Neurotrophic Factor -- physiology
- Cell Line, Tumor
- Cisplatin -- therapeutic use
- Drug Resistance, Neoplasm
- Etoposide -- pharmacology
- Flow Cytometry
- Glycogen Synthase Kinase 3 -- antagonists & inhibitors
- Glycogen Synthase Kinase 3 beta
- Humans
- Membrane Potentials -- drug effects
- Neuroblastoma -- drug therapy
- Phosphorylation
- Proto-Oncogene Proteins c-akt -- metabolism
- RNA, Small Interfering
- Receptor, trkB -- physiology
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Publication Type: Journal Article; Research Support, N.I.H., Intramural
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