Inactivation of glycogen synthase kinase-3beta contributes to brain-derived neutrophic factor/TrkB-induced resistance to chemotherapy in neuroblastoma cells.
Li, Zhijie
Inactivation of glycogen synthase kinase-3beta contributes to brain-derived neutrophic factor/TrkB-induced resistance to chemotherapy in neuroblastoma cells. [electronic resource] - Molecular cancer therapeutics Dec 2007 - 3113-21 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Intramural
1535-7163
10.1158/1535-7163.MCT-07-0133 doi
Antineoplastic Agents--therapeutic use
Brain-Derived Neurotrophic Factor--physiology
Cell Line, Tumor
Cisplatin--therapeutic use
Drug Resistance, Neoplasm
Etoposide--pharmacology
Flow Cytometry
Glycogen Synthase Kinase 3--antagonists & inhibitors
Glycogen Synthase Kinase 3 beta
Humans
Membrane Potentials--drug effects
Neuroblastoma--drug therapy
Phosphorylation
Proto-Oncogene Proteins c-akt--metabolism
RNA, Small Interfering
Receptor, trkB--physiology
Inactivation of glycogen synthase kinase-3beta contributes to brain-derived neutrophic factor/TrkB-induced resistance to chemotherapy in neuroblastoma cells. [electronic resource] - Molecular cancer therapeutics Dec 2007 - 3113-21 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Intramural
1535-7163
10.1158/1535-7163.MCT-07-0133 doi
Antineoplastic Agents--therapeutic use
Brain-Derived Neurotrophic Factor--physiology
Cell Line, Tumor
Cisplatin--therapeutic use
Drug Resistance, Neoplasm
Etoposide--pharmacology
Flow Cytometry
Glycogen Synthase Kinase 3--antagonists & inhibitors
Glycogen Synthase Kinase 3 beta
Humans
Membrane Potentials--drug effects
Neuroblastoma--drug therapy
Phosphorylation
Proto-Oncogene Proteins c-akt--metabolism
RNA, Small Interfering
Receptor, trkB--physiology