Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 pathway. [electronic resource]
Producer: 20070830Description: 535-44 p. digitalISSN:- 1524-4539
- Adaptation, Physiological -- physiology
- Animals
- Cyclooxygenase 2 -- biosynthesis
- DNA -- genetics
- Enzyme Activation -- drug effects
- Enzyme Induction -- drug effects
- Hypertension -- complications
- Ischemic Preconditioning, Myocardial
- MAP Kinase Kinase Kinase 1 -- physiology
- MAP Kinase Kinase Kinase 2 -- physiology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Mutant Strains
- Mitogen-Activated Protein Kinase 1 -- physiology
- Mitogen-Activated Protein Kinase 3 -- physiology
- Myocardial Infarction -- pathology
- Myocardial Reperfusion Injury -- physiopathology
- Nitric Oxide -- physiology
- Nitric Oxide Donors -- pharmacology
- Nitric Oxide Synthase Type II -- deficiency
- Nitric Oxide Synthase Type III
- Phosphorylation -- drug effects
- Protein Kinase C-epsilon -- physiology
- Protein Processing, Post-Translational -- drug effects
- STAT1 Transcription Factor -- physiology
- STAT3 Transcription Factor -- physiology
- Signal Transduction -- drug effects
- Specific Pathogen-Free Organisms
- Stress, Physiological -- physiopathology
- Time Factors
- Triazenes -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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