Thyrocytes responding to IFN-gamma are essential for development of lymphocytic spontaneous autoimmune thyroiditis and inhibition of thyrocyte hyperplasia. [electronic resource]
Producer: 20060309Description: 1259-65 p. digitalISSN:- 0022-1767
- Adoptive Transfer
- Animals
- Base Sequence
- Chemokines -- genetics
- Hyperplasia
- Interferon-gamma -- deficiency
- Lymphocytes -- immunology
- Male
- Mice
- Mice, Inbred NOD
- Mice, Knockout
- RNA, Messenger -- genetics
- Receptors, Interferon -- deficiency
- Thyroid Gland -- immunology
- Thyroiditis, Autoimmune -- etiology
- Interferon gamma Receptor
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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