Thyrocytes responding to IFN-gamma are essential for development of lymphocytic spontaneous autoimmune thyroiditis and inhibition of thyrocyte hyperplasia.

Yu, Shiguang

Thyrocytes responding to IFN-gamma are essential for development of lymphocytic spontaneous autoimmune thyroiditis and inhibition of thyrocyte hyperplasia. [electronic resource] - Journal of immunology (Baltimore, Md. : 1950) Jan 2006 - 1259-65 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.

0022-1767

10.4049/jimmunol.176.2.1259 doi


Adoptive Transfer
Animals
Base Sequence
Chemokines--genetics
Hyperplasia
Interferon-gamma--deficiency
Lymphocytes--immunology
Male
Mice
Mice, Inbred NOD
Mice, Knockout
RNA, Messenger--genetics
Receptors, Interferon--deficiency
Thyroid Gland--immunology
Thyroiditis, Autoimmune--etiology
Interferon gamma Receptor