Isoflurane inhibits cardiac myocyte apoptosis during oxidative and inflammatory stress by activating Akt and enhancing Bcl-2 expression. [electronic resource]
Producer: 20060125Description: 1006-14 p. digitalISSN:- 0003-3022
- Anesthetics, Inhalation -- pharmacology
- Animals
- Animals, Newborn
- Apoptosis -- drug effects
- Biotransformation -- drug effects
- Caspases -- metabolism
- Cell Line
- Cell Separation
- Cytochromes c -- metabolism
- Dogs
- Genes, bcl-2 -- drug effects
- Hydrogen Peroxide -- pharmacology
- Hypoxia -- metabolism
- In Situ Nick-End Labeling
- Inflammation -- pathology
- Isoflurane -- pharmacology
- L-Lactate Dehydrogenase -- metabolism
- Male
- Myocytes, Cardiac -- drug effects
- Neutrophil Activation -- drug effects
- Neutrophils -- drug effects
- Oxidation-Reduction
- Oxidative Stress -- drug effects
- Proto-Oncogene Proteins c-akt -- biosynthesis
- Rats
- Rats, Sprague-Dawley
- Stimulation, Chemical
- Tetrazolium Salts -- metabolism
- Thiazoles -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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