Isoflurane inhibits cardiac myocyte apoptosis during oxidative and inflammatory stress by activating Akt and enhancing Bcl-2 expression.

Jamnicki-Abegg, Marina

Isoflurane inhibits cardiac myocyte apoptosis during oxidative and inflammatory stress by activating Akt and enhancing Bcl-2 expression. [electronic resource] - Anesthesiology Nov 2005 - 1006-14 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

0003-3022

10.1097/00000542-200511000-00015 doi


Anesthetics, Inhalation--pharmacology
Animals
Animals, Newborn
Apoptosis--drug effects
Biotransformation--drug effects
Caspases--metabolism
Cell Line
Cell Separation
Cytochromes c--metabolism
Dogs
Genes, bcl-2--drug effects
Hydrogen Peroxide--pharmacology
Hypoxia--metabolism
In Situ Nick-End Labeling
Inflammation--pathology
Isoflurane--pharmacology
L-Lactate Dehydrogenase--metabolism
Male
Myocytes, Cardiac--drug effects
Neutrophil Activation--drug effects
Neutrophils--drug effects
Oxidation-Reduction
Oxidative Stress--drug effects
Proto-Oncogene Proteins c-akt--biosynthesis
Rats
Rats, Sprague-Dawley
Stimulation, Chemical
Tetrazolium Salts--metabolism
Thiazoles--metabolism