Prevention of cytosolic IAPs degradation: a potential pharmacological target in Huntington's Disease. [electronic resource]
Producer: 20050930Description: 140-50 p. digitalISSN:- 1043-6618
- Animals
- Apoptosis Regulatory Proteins
- Brain -- metabolism
- Carrier Proteins -- metabolism
- Cell Line
- Cell Survival
- Cyclosporine -- pharmacology
- Cytosol -- metabolism
- High-Temperature Requirement A Serine Peptidase 2
- Humans
- Huntingtin Protein
- Huntington Disease -- genetics
- Inhibitor of Apoptosis Proteins
- Intracellular Signaling Peptides and Proteins
- Mice
- Mitochondria -- metabolism
- Mitochondrial Proteins -- metabolism
- Mutation
- Nerve Tissue Proteins -- genetics
- Nuclear Proteins -- genetics
- Proteins -- metabolism
- Pyrimidinones -- pharmacology
- Serine Endopeptidases -- metabolism
- Thiones -- pharmacology
- Ubiquitin-Protein Ligases
- X-Linked Inhibitor of Apoptosis Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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