Neuroprotection against focal ischemic brain injury by inhibition of c-Jun N-terminal kinase and attenuation of the mitochondrial apoptosis-signaling pathway. [electronic resource]
Producer: 20050621Description: 694-712 p. digitalISSN:- 0271-678X
- 14-3-3 Proteins -- metabolism
- Animals
- Anthracenes -- pharmacology
- Apoptosis -- drug effects
- Apoptosis Regulatory Proteins
- Bcl-2-Like Protein 11
- Brain Ischemia -- drug therapy
- Carrier Proteins -- metabolism
- Cerebral Infarction -- drug therapy
- Enzyme Inhibitors -- pharmacology
- JNK Mitogen-Activated Protein Kinases -- antagonists & inhibitors
- Membrane Proteins -- metabolism
- Mice
- Mice, Inbred C57BL
- Mitochondria -- drug effects
- Neuroprotective Agents -- pharmacology
- Phosphorylation
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Reperfusion Injury -- drug therapy
- Signal Transduction -- drug effects
- bcl-2-Associated X Protein
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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