Neuroprotection against focal ischemic brain injury by inhibition of c-Jun N-terminal kinase and attenuation of the mitochondrial apoptosis-signaling pathway.

Gao, Yanqin

Neuroprotection against focal ischemic brain injury by inhibition of c-Jun N-terminal kinase and attenuation of the mitochondrial apoptosis-signaling pathway. [electronic resource] - Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism Jun 2005 - 694-712 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0271-678X

10.1038/sj.jcbfm.9600062 doi


14-3-3 Proteins--metabolism
Animals
Anthracenes--pharmacology
Apoptosis--drug effects
Apoptosis Regulatory Proteins
Bcl-2-Like Protein 11
Brain Ischemia--drug therapy
Carrier Proteins--metabolism
Cerebral Infarction--drug therapy
Enzyme Inhibitors--pharmacology
JNK Mitogen-Activated Protein Kinases--antagonists & inhibitors
Membrane Proteins--metabolism
Mice
Mice, Inbred C57BL
Mitochondria--drug effects
Neuroprotective Agents--pharmacology
Phosphorylation
Proto-Oncogene Proteins--metabolism
Proto-Oncogene Proteins c-bcl-2--metabolism
Reperfusion Injury--drug therapy
Signal Transduction--drug effects
bcl-2-Associated X Protein