Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor. [electronic resource]
Producer: 20050531Description: H2093-101 p. digitalISSN:- 0363-6135
- Adenosine Triphosphate -- metabolism
- Animals
- Cell Survival -- drug effects
- Epoprostenol -- analogs & derivatives
- Flavoproteins -- metabolism
- Fluorescence
- Hydrogen Peroxide -- pharmacology
- Mitochondria -- drug effects
- Myocytes, Cardiac -- drug effects
- Oxidants -- pharmacology
- Oxidative Stress -- drug effects
- Potassium Channels -- metabolism
- RNA, Messenger -- analysis
- Rats
- Rats, Sprague-Dawley
- Receptors, Prostaglandin E -- genetics
- Receptors, Prostaglandin E, EP3 Subtype
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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