Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.

Shinmura, Ken

Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor. [electronic resource] - American journal of physiology. Heart and circulatory physiology May 2005 - H2093-101 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0363-6135

10.1152/ajpheart.01003.2004 doi


Adenosine Triphosphate--metabolism
Animals
Cell Survival--drug effects
Epoprostenol--analogs & derivatives
Flavoproteins--metabolism
Fluorescence
Hydrogen Peroxide--pharmacology
Mitochondria--drug effects
Myocytes, Cardiac--drug effects
Oxidants--pharmacology
Oxidative Stress--drug effects
Potassium Channels--metabolism
RNA, Messenger--analysis
Rats
Rats, Sprague-Dawley
Receptors, Prostaglandin E--genetics
Receptors, Prostaglandin E, EP3 Subtype