Cathepsin-dependent apoptosis triggered by supraoptimal activation of T lymphocytes: a possible mechanism of high dose tolerance. [electronic resource]
Producer: 20040817Description: 5405-14 p. digitalISSN:- 0022-1767
- Amino Acid Chloromethyl Ketones -- pharmacology
- Apoptosis -- immunology
- CD28 Antigens -- pharmacology
- Caspase Inhibitors
- Catalysis
- Cathepsin B -- antagonists & inhibitors
- Cathepsin L
- Cathepsins -- antagonists & inhibitors
- Cell Death -- immunology
- Cell Differentiation -- immunology
- Cells, Cultured
- Cysteine Endopeptidases
- Cysteine Proteinase Inhibitors -- pharmacology
- Cytochromes c -- metabolism
- Cytosol -- enzymology
- DNA Fragmentation -- drug effects
- Dose-Response Relationship, Immunologic
- G1 Phase -- immunology
- Humans
- Intracellular Membranes -- enzymology
- Lymphocyte Activation -- immunology
- Lysosomes -- enzymology
- Muromonab-CD3 -- pharmacology
- Permeability
- S Phase -- immunology
- T-Lymphocytes -- cytology
- Tetradecanoylphorbol Acetate -- pharmacology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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