Homocysteine increases the expression of vascular endothelial growth factor by a mechanism involving endoplasmic reticulum stress and transcription factor ATF4. [electronic resource]
Producer: 20040601Description: 14844-52 p. digitalISSN:- 0021-9258
- Activating Transcription Factor 4
- Blotting, Northern
- Blotting, Western
- CCAAT-Enhancer-Binding Proteins -- metabolism
- Carrier Proteins -- metabolism
- Cell Line
- Cell Nucleus -- metabolism
- DNA, Complementary -- metabolism
- Dactinomycin -- pharmacology
- Dose-Response Relationship, Drug
- Endoplasmic Reticulum -- metabolism
- Endoplasmic Reticulum Chaperone BiP
- Enzyme-Linked Immunosorbent Assay
- Epithelial Cells -- cytology
- Genes, Dominant
- Heat-Shock Proteins
- Homocysteine -- chemistry
- Humans
- Molecular Chaperones
- Mutation
- Neovascularization, Pathologic
- Nucleic Acid Synthesis Inhibitors -- pharmacology
- Phosphorylation
- Promoter Regions, Genetic
- Protein Binding
- RNA, Messenger -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Stress, Physiological
- Time Factors
- Transcription Factor CHOP
- Transcription Factors -- metabolism
- Transcription, Genetic
- Up-Regulation
- Vascular Endothelial Growth Factor A -- biosynthesis
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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