Retinoic acids exert direct effects on T cells to suppress Th1 development and enhance Th2 development via retinoic acid receptors. [electronic resource]
Producer: 20040505Description: 1017-25 p. digitalISSN:- 0953-8178
- Alitretinoin
- Animals
- Antibodies -- pharmacology
- CD28 Antigens -- immunology
- CD3 Complex -- immunology
- Cell Differentiation -- drug effects
- DNA-Binding Proteins -- genetics
- Dose-Response Relationship, Drug
- Flow Cytometry
- GATA3 Transcription Factor
- Gene Expression Regulation -- drug effects
- Histocompatibility Antigens -- genetics
- Interferon-gamma -- metabolism
- Interleukin-10 -- metabolism
- Interleukin-12 -- pharmacology
- Interleukin-2 -- pharmacology
- Interleukin-4 -- metabolism
- Interleukin-5 -- metabolism
- Lymphocyte Activation -- drug effects
- Male
- Mice
- Mice, Knockout
- Mice, Transgenic
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins c-maf
- Receptors, Interleukin -- genetics
- Receptors, Interleukin-12
- Receptors, Interleukin-4 -- genetics
- Receptors, Retinoic Acid -- agonists
- Retinoid X Receptors
- Reverse Transcriptase Polymerase Chain Reaction
- T-Box Domain Proteins
- T-Lymphocytes -- cytology
- Th1 Cells -- drug effects
- Th2 Cells -- drug effects
- Time Factors
- Trans-Activators -- genetics
- Transcription Factors -- agonists
- Tretinoin -- pharmacology
- Vitamin A -- pharmacology
- T-bet Transcription Factor
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Publication Type: Journal Article
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