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Iwata, Makoto

Retinoic acids exert direct effects on T cells to suppress Th1 development and enhance Th2 development via retinoic acid receptors. [electronic resource] - International immunology Aug 2003 - 1017-25 p. digital

Publication Type: Journal Article

ISSN: 0953-8178

Standard No.: 10.1093/intimm/dxg101 doi

Subjects--Topical Terms:
Alitretinoin
Animals
Antibodies--pharmacology
CD28 Antigens--immunology
CD3 Complex--immunology
Cell Differentiation--drug effects
DNA-Binding Proteins--genetics
Dose-Response Relationship, Drug
Flow Cytometry
GATA3 Transcription Factor
Gene Expression Regulation--drug effects
Histocompatibility Antigens--genetics
Interferon-gamma--metabolism
Interleukin-10--metabolism
Interleukin-12--pharmacology
Interleukin-2--pharmacology
Interleukin-4--metabolism
Interleukin-5--metabolism
Lymphocyte Activation--drug effects
Male
Mice
Mice, Knockout
Mice, Transgenic
Proto-Oncogene Proteins--genetics
Proto-Oncogene Proteins c-maf
Receptors, Interleukin--genetics
Receptors, Interleukin-12
Receptors, Interleukin-4--genetics
Receptors, Retinoic Acid--agonists
Retinoid X Receptors
Reverse Transcriptase Polymerase Chain Reaction
T-Box Domain Proteins
T-Lymphocytes--cytology
Th1 Cells--drug effects
Th2 Cells--drug effects
Time Factors
Trans-Activators--genetics
Transcription Factors--agonists
Tretinoin--pharmacology
Vitamin A--pharmacology
T-bet Transcription Factor