Paralytic autoimmune myositis develops in nonobese diabetic mice made Th1 cytokine-deficient by expression of an IFN-gamma receptor beta-chain transgene. [electronic resource]
Producer: 20030521Description: 2742-9 p. digitalISSN:- 0022-1767
- Adoptive Transfer
- Animals
- CD8-Positive T-Lymphocytes -- immunology
- Cell Movement -- genetics
- Cytokines -- deficiency
- Diabetes Mellitus, Type 1 -- genetics
- Female
- Gene Expression Regulation -- immunology
- Interferon-gamma -- antagonists & inhibitors
- Kinetics
- Macrophages -- pathology
- Mice
- Mice, Inbred C57BL
- Mice, Inbred NOD
- Mice, SCID
- Mice, Transgenic
- Nervous System Autoimmune Disease, Experimental -- genetics
- Paralysis -- genetics
- Receptors, Interferon -- biosynthesis
- Sex Characteristics
- Spleen -- immunology
- Th1 Cells -- immunology
- Transgenes -- immunology
- Tumor Necrosis Factor-alpha -- antagonists & inhibitors
- Interferon gamma Receptor
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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