Regulation of the IRF-1 tumour modifier during the response to genotoxic stress involves an ATM-dependent signalling pathway. [electronic resource]
Producer: 20021212Description: 7776-85 p. digitalISSN:- 0950-9232
- Androstadienes -- pharmacology
- Ataxia Telangiectasia -- genetics
- Ataxia Telangiectasia Mutated Proteins
- Cell Cycle Proteins
- Cells, Cultured -- metabolism
- Cyclin-Dependent Kinase Inhibitor p21
- Cyclins -- physiology
- Cycloheximide -- pharmacology
- DNA -- drug effects
- DNA Damage
- DNA-Binding Proteins -- biosynthesis
- Enzyme Inhibitors -- pharmacology
- Etoposide -- toxicity
- Fibroblasts -- metabolism
- Gamma Rays -- adverse effects
- Genes, Reporter
- Genes, p53
- Humans
- Interferon Regulatory Factor-1
- Melanoma -- pathology
- Phosphatidylinositol 3-Kinases -- physiology
- Phosphoinositide-3 Kinase Inhibitors
- Phosphoproteins -- biosynthesis
- Phosphorylation
- Poly I-C -- pharmacology
- Protein Processing, Post-Translational
- Protein Serine-Threonine Kinases -- deficiency
- Protein Synthesis Inhibitors -- pharmacology
- RNA, Messenger -- biosynthesis
- Recombinant Fusion Proteins -- biosynthesis
- Signal Transduction -- physiology
- Transcription, Genetic
- Tumor Cells, Cultured -- metabolism
- Tumor Suppressor Protein p53 -- biosynthesis
- Tumor Suppressor Proteins
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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