Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment. [electronic resource]
Producer: 20020219Description: 65-77 p. digitalISSN:- 0950-9232
- Amino Acid Chloromethyl Ketones -- pharmacology
- Apoptosis -- drug effects
- Apoptosis Inducing Factor
- Carcinoma, Non-Small-Cell Lung -- pathology
- Carcinoma, Small Cell -- pathology
- Caspase 3
- Caspase 7
- Caspases -- biosynthesis
- Cell Cycle -- drug effects
- Cell Nucleus -- drug effects
- Cysteine Proteinase Inhibitors -- pharmacology
- Cytochrome c Group -- analysis
- Drug Resistance, Neoplasm
- Enzyme Activation -- drug effects
- Enzyme Inhibitors -- pharmacology
- Enzyme Precursors -- biosynthesis
- Flavoproteins -- physiology
- Gamma Rays
- Humans
- Jurkat Cells -- drug effects
- Lung Neoplasms -- pathology
- Membrane Potentials -- drug effects
- Membrane Proteins -- physiology
- Mitochondria -- physiology
- Poly(ADP-ribose) Polymerases -- metabolism
- Protein Transport
- Radiation Tolerance
- Reactive Oxygen Species -- metabolism
- Recombinant Fusion Proteins -- physiology
- Staurosporine -- pharmacology
- Superoxides -- metabolism
- Tumor Cells, Cultured -- drug effects
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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