PR-39 and PR-11 peptides inhibit ischemia-reperfusion injury by blocking proteasome-mediated I kappa B alpha degradation. [electronic resource]
Producer: 20020107Description: H2612-8 p. digitalISSN:- 0363-6135
- Animals
- Anti-Bacterial Agents -- pharmacology
- Antimicrobial Cationic Peptides -- pharmacology
- Cells, Cultured
- Cysteine Endopeptidases -- metabolism
- DNA-Binding Proteins -- metabolism
- Endothelium, Vascular -- cytology
- Humans
- I-kappa B Proteins
- Intercellular Adhesion Molecule-1 -- metabolism
- Male
- Multienzyme Complexes -- metabolism
- Myocardial Reperfusion Injury -- drug therapy
- Myocardium -- metabolism
- NADPH Oxidases -- metabolism
- NF-KappaB Inhibitor alpha
- Neutrophils -- metabolism
- Peptide Fragments -- pharmacology
- Peroxidase -- metabolism
- Phosphoproteins -- metabolism
- Proteasome Endopeptidase Complex
- Rats
- Rats, Sprague-Dawley
- Reactive Oxygen Species -- metabolism
- Umbilical Veins -- cytology
- Vascular Cell Adhesion Molecule-1 -- pharmacology
- Ventricular Function, Left -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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