Normal view MARC view ISBD view

Bao, J

PR-39 and PR-11 peptides inhibit ischemia-reperfusion injury by blocking proteasome-mediated I kappa B alpha degradation. [electronic resource] - American journal of physiology. Heart and circulatory physiology Dec 2001 - H2612-8 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

ISSN: 0363-6135

Standard No.: 10.1152/ajpheart.2001.281.6.H2612 doi

Subjects--Topical Terms:
Animals
Anti-Bacterial Agents--pharmacology
Antimicrobial Cationic Peptides--pharmacology
Cells, Cultured
Cysteine Endopeptidases--metabolism
DNA-Binding Proteins--metabolism
Endothelium, Vascular--cytology
Humans
I-kappa B Proteins
Intercellular Adhesion Molecule-1--metabolism
Male
Multienzyme Complexes--metabolism
Myocardial Reperfusion Injury--drug therapy
Myocardium--metabolism
NADPH Oxidases--metabolism
NF-KappaB Inhibitor alpha
Neutrophils--metabolism
Peptide Fragments--pharmacology
Peroxidase--metabolism
Phosphoproteins--metabolism
Proteasome Endopeptidase Complex
Rats
Rats, Sprague-Dawley
Reactive Oxygen Species--metabolism
Umbilical Veins--cytology
Vascular Cell Adhesion Molecule-1--pharmacology
Ventricular Function, Left--drug effects