Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function. [electronic resource]
Producer: 20010913Description: 438-45 p. digitalISSN:- 1469-221X
- Actins -- metabolism
- Animals
- Apoptosis
- Blood Vessels -- embryology
- Embryonic and Fetal Development
- Endothelium, Vascular -- cytology
- Heart -- embryology
- Hemorrhage
- Immunohistochemistry
- In Situ Nick-End Labeling
- Mice
- Mice, Transgenic
- Neovascularization, Physiologic
- Receptor Protein-Tyrosine Kinases -- genetics
- Receptor, TIE-2
- Signal Transduction
- Transgenes
- von Willebrand Factor -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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