Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function.
Jones, N
Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function. [electronic resource] - EMBO reports May 2001 - 438-45 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1469-221X
10.1093/embo-reports/kve093 doi
Actins--metabolism
Animals
Apoptosis
Blood Vessels--embryology
Embryonic and Fetal Development
Endothelium, Vascular--cytology
Heart--embryology
Hemorrhage
Immunohistochemistry
In Situ Nick-End Labeling
Mice
Mice, Transgenic
Neovascularization, Physiologic
Receptor Protein-Tyrosine Kinases--genetics
Receptor, TIE-2
Signal Transduction
Transgenes
von Willebrand Factor--metabolism
Rescue of the early vascular defects in Tek/Tie2 null mice reveals an essential survival function. [electronic resource] - EMBO reports May 2001 - 438-45 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1469-221X
10.1093/embo-reports/kve093 doi
Actins--metabolism
Animals
Apoptosis
Blood Vessels--embryology
Embryonic and Fetal Development
Endothelium, Vascular--cytology
Heart--embryology
Hemorrhage
Immunohistochemistry
In Situ Nick-End Labeling
Mice
Mice, Transgenic
Neovascularization, Physiologic
Receptor Protein-Tyrosine Kinases--genetics
Receptor, TIE-2
Signal Transduction
Transgenes
von Willebrand Factor--metabolism