Western and Chinese antirheumatic drug-induced T cell apoptotic DNA damage uses different caspase cascades and is independent of Fas/Fas ligand interaction. [electronic resource]
Producer: 20010816Description: 6914-24 p. digitalISSN:- 0022-1767
- Alkaloids -- toxicity
- Anti-Inflammatory Agents, Non-Steroidal -- toxicity
- Apoptosis -- drug effects
- Benzylisoquinolines
- Caspase 3
- Caspase 8
- Caspase 9
- Caspase Inhibitors
- Caspases -- biosynthesis
- Cell Death -- drug effects
- Cysteine Proteinase Inhibitors -- pharmacology
- DNA Damage -- immunology
- Drugs, Chinese Herbal -- toxicity
- Enzyme Activation -- drug effects
- Enzyme Induction -- drug effects
- Fas Ligand Protein
- Humans
- Hydroxychloroquine -- toxicity
- Immunity, Innate -- drug effects
- Jurkat Cells -- cytology
- Lymphocyte Activation -- drug effects
- Lymphocyte Count
- Membrane Glycoproteins -- biosynthesis
- Methotrexate -- toxicity
- Signal Transduction -- drug effects
- T-Lymphocyte Subsets -- cytology
- Tripterygium
- U937 Cells
- fas Receptor -- biosynthesis
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Publication Type: Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
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