NF-kappa B RelA (p65) is essential for TNF-alpha-induced fas expression but dispensable for both TCR-induced expression and activation-induced cell death. [electronic resource]
Producer: 20010628Description: 4949-57 p. digitalISSN:- 0022-1767
- 3T3 Cells
- Animals
- Apoptosis -- drug effects
- Base Sequence
- Binding Sites -- genetics
- CD4-Positive T-Lymphocytes -- cytology
- Cells, Cultured
- Cloning, Molecular
- Down-Regulation -- genetics
- Fas Ligand Protein
- Fibroblasts -- metabolism
- Humans
- Jurkat Cells
- Ligands
- Lymphocyte Activation -- drug effects
- Membrane Glycoproteins -- biosynthesis
- Mice
- Mice, Inbred C57BL
- Molecular Sequence Data
- NF-kappa B -- deficiency
- NF-kappa B p50 Subunit
- Phytohemagglutinins -- pharmacology
- Promoter Regions, Genetic -- immunology
- Proto-Oncogene Proteins c-rel -- deficiency
- Receptors, Antigen, T-Cell -- physiology
- Tetradecanoylphorbol Acetate -- pharmacology
- Transcription Factor RelA
- Tumor Necrosis Factor-alpha -- physiology
- fas Receptor -- biosynthesis
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Publication Type: Journal Article; Research Support, U.S. Gov't, P.H.S.
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