A switch from p130Cas/Crk to Gab1/Crk signaling correlates with anchorage independent growth and JNK activation in cells transformed by the Met receptor oncoprotein. [electronic resource]
Producer: 20010201Description: 5973-81 p. digitalISSN:- 0950-9232
- 3T3 Cells
- Actins -- metabolism
- Adaptor Proteins, Signal Transducing
- Animals
- Cell Adhesion
- Cell Division
- Cell Size
- Cell Transformation, Neoplastic -- metabolism
- Crk-Associated Substrate Protein
- Cytoskeletal Proteins -- metabolism
- Enzyme Activation
- Fibronectins -- metabolism
- Focal Adhesion Kinase 1
- Focal Adhesion Protein-Tyrosine Kinases
- Focal Adhesions -- metabolism
- JNK Mitogen-Activated Protein Kinases
- MAP Kinase Kinase 4
- Mice
- Mitogen-Activated Protein Kinase Kinases -- metabolism
- Mutation -- genetics
- Oncogene Proteins -- genetics
- Paxillin
- Phosphoproteins -- metabolism
- Phosphorylation
- Phosphotyrosine -- metabolism
- Protein Binding
- Protein-Tyrosine Kinases -- metabolism
- Proteins
- Proto-Oncogene Proteins -- metabolism
- Proto-Oncogene Proteins c-crk
- Proto-Oncogene Proteins c-met -- genetics
- Retinoblastoma-Like Protein p130
- Signal Transduction
- Stress Fibers -- metabolism
- Wound Healing
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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