Overexpression of the wild-type p53 gene inhibits NF-kappaB activity and synergizes with aspirin to induce apoptosis in human colon cancer cells. [electronic resource]
Producer: 20000316Description: 726-36 p. digitalISSN:- 0950-9232
- Adenocarcinoma -- genetics
- Adenoviruses, Human -- genetics
- Anti-Inflammatory Agents, Non-Steroidal -- pharmacology
- Apoptosis -- drug effects
- Aspirin -- pharmacology
- Cell Nucleus -- metabolism
- Colonic Neoplasms -- genetics
- Cytomegalovirus -- genetics
- DNA-Binding Proteins -- biosynthesis
- Gene Expression Regulation -- drug effects
- Genes, p53
- Genetic Vectors -- genetics
- Humans
- I-kappa B Proteins
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Neoplasm Proteins -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Transcription, Genetic -- drug effects
- Tumor Cells, Cultured
- Tumor Suppressor Protein p53 -- biosynthesis
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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