DNA base excision repair in human malaria parasites is predominantly by a long-patch pathway. [electronic resource]
Producer: 20000223Description: 763-72 p. digitalISSN:- 0006-2960
- Animals
- Binding Sites -- genetics
- Carbon-Oxygen Lyases -- metabolism
- Cell-Free System -- enzymology
- DNA Glycosylases
- DNA Repair
- DNA, Circular -- metabolism
- DNA, Protozoan -- metabolism
- DNA-(Apurinic or Apyrimidinic Site) Lyase
- Deoxyribonuclease IV (Phage T4-Induced)
- Endodeoxyribonucleases -- metabolism
- Enzyme Activation
- Escherichia coli Proteins
- Flap Endonucleases
- Humans
- Malaria, Falciparum -- enzymology
- N-Glycosyl Hydrolases -- metabolism
- Plasmids -- metabolism
- Plasmodium falciparum -- enzymology
- Uracil-DNA Glycosidase
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
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