DNA base excision repair in human malaria parasites is predominantly by a long-patch pathway.

Haltiwanger, B M

DNA base excision repair in human malaria parasites is predominantly by a long-patch pathway. [electronic resource] - Biochemistry Feb 2000 - 763-72 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.

0006-2960

10.1021/bi9923151 doi


Animals
Binding Sites--genetics
Carbon-Oxygen Lyases--metabolism
Cell-Free System--enzymology
DNA Glycosylases
DNA Repair
DNA, Circular--metabolism
DNA, Protozoan--metabolism
DNA-(Apurinic or Apyrimidinic Site) Lyase
Deoxyribonuclease IV (Phage T4-Induced)
Endodeoxyribonucleases--metabolism
Enzyme Activation
Escherichia coli Proteins
Flap Endonucleases
Humans
Malaria, Falciparum--enzymology
N-Glycosyl Hydrolases--metabolism
Plasmids--metabolism
Plasmodium falciparum--enzymology
Uracil-DNA Glycosidase